node1 | node2 | node1 accession | node2 accession | node1 annotation | node2 annotation | score |
AEA67223.1 | AEA67248.1 | PSEBR_a1050 | PSEBR_a1074 | Conserved hypothetical protein. | Putative outer membrane chaperone Skp; putative exported protein. | 0.752 |
AEA67223.1 | AEA67963.1 | PSEBR_a1050 | PSEBR_a1752 | Conserved hypothetical protein. | Putative cell division inhibitor; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | 0.628 |
AEA67223.1 | AEA67965.1 | PSEBR_a1050 | PSEBR_a1754 | Conserved hypothetical protein. | Transcriptional regulator. | 0.485 |
AEA67223.1 | AEA70878.1 | PSEBR_a1050 | PSEBR_a4467 | Conserved hypothetical protein. | Putative DNA binding protein MvaT, H-NS family. | 0.747 |
AEA67248.1 | AEA67223.1 | PSEBR_a1074 | PSEBR_a1050 | Putative outer membrane chaperone Skp; putative exported protein. | Conserved hypothetical protein. | 0.752 |
AEA67248.1 | AEA67965.1 | PSEBR_a1074 | PSEBR_a1754 | Putative outer membrane chaperone Skp; putative exported protein. | Transcriptional regulator. | 0.484 |
AEA67248.1 | AEA70878.1 | PSEBR_a1074 | PSEBR_a4467 | Putative outer membrane chaperone Skp; putative exported protein. | Putative DNA binding protein MvaT, H-NS family. | 0.721 |
AEA67931.1 | AEA67965.1 | PSEBR_a1722 | PSEBR_a1754 | Conserved hypothetical protein. | Transcriptional regulator. | 0.449 |
AEA67963.1 | AEA67223.1 | PSEBR_a1752 | PSEBR_a1050 | Putative cell division inhibitor; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | Conserved hypothetical protein. | 0.628 |
AEA67963.1 | AEA67965.1 | PSEBR_a1752 | PSEBR_a1754 | Putative cell division inhibitor; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | Transcriptional regulator. | 0.546 |
AEA67963.1 | lexA | PSEBR_a1752 | PSEBR_a1753 | Putative cell division inhibitor; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | Putative transcription factor, LexA family; Represses a number of genes involved in the response to DNA damage (SOS response), including recA and lexA. In the presence of single-stranded DNA, RecA interacts with LexA causing an autocatalytic cleavage which disrupts the DNA-binding part of LexA, leading to derepression of the SOS regulon and eventually DNA repair. | 0.905 |
AEA67963.1 | nagZ | PSEBR_a1752 | PSEBR_a1755 | Putative cell division inhibitor; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | Beta-N-acetylhexosaminidase (beta-hexosaminidase); Plays a role in peptidoglycan recycling by cleaving the terminal beta-1,4-linked N-acetylglucosamine (GlcNAc) from peptide- linked peptidoglycan fragments, giving rise to free GlcNAc, anhydro-N- acetylmuramic acid and anhydro-N-acetylmuramic acid-linked peptides. Belongs to the glycosyl hydrolase 3 family. NagZ subfamily. | 0.413 |
AEA67965.1 | AEA67223.1 | PSEBR_a1754 | PSEBR_a1050 | Transcriptional regulator. | Conserved hypothetical protein. | 0.485 |
AEA67965.1 | AEA67248.1 | PSEBR_a1754 | PSEBR_a1074 | Transcriptional regulator. | Putative outer membrane chaperone Skp; putative exported protein. | 0.484 |
AEA67965.1 | AEA67931.1 | PSEBR_a1754 | PSEBR_a1722 | Transcriptional regulator. | Conserved hypothetical protein. | 0.449 |
AEA67965.1 | AEA67963.1 | PSEBR_a1754 | PSEBR_a1752 | Transcriptional regulator. | Putative cell division inhibitor; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | 0.546 |
AEA67965.1 | AEA68671.1 | PSEBR_a1754 | PSEBR_a2391 | Transcriptional regulator. | Putative peptide synthase. | 0.411 |
AEA67965.1 | AEA70878.1 | PSEBR_a1754 | PSEBR_a4467 | Transcriptional regulator. | Putative DNA binding protein MvaT, H-NS family. | 0.456 |
AEA67965.1 | lexA | PSEBR_a1754 | PSEBR_a1753 | Transcriptional regulator. | Putative transcription factor, LexA family; Represses a number of genes involved in the response to DNA damage (SOS response), including recA and lexA. In the presence of single-stranded DNA, RecA interacts with LexA causing an autocatalytic cleavage which disrupts the DNA-binding part of LexA, leading to derepression of the SOS regulon and eventually DNA repair. | 0.554 |
AEA67965.1 | nagZ | PSEBR_a1754 | PSEBR_a1755 | Transcriptional regulator. | Beta-N-acetylhexosaminidase (beta-hexosaminidase); Plays a role in peptidoglycan recycling by cleaving the terminal beta-1,4-linked N-acetylglucosamine (GlcNAc) from peptide- linked peptidoglycan fragments, giving rise to free GlcNAc, anhydro-N- acetylmuramic acid and anhydro-N-acetylmuramic acid-linked peptides. Belongs to the glycosyl hydrolase 3 family. NagZ subfamily. | 0.554 |